REASON FOR THE RESEARCH

Due to the loss of a fiancé and some friends to Mr. Duda; the fact that Mr.McDonald's father had cancer, which was removed, and the loss of his father-in-law to cancer, prompted this research.

ORIGINAL HYPOTHESIS

A high concentration of some chemical element in the body was causing the cells to fail to cease reproduction in the normal way.

ORIGINAL FINDINGS

The blood type inside the tumor mass is different than that of the patient's primary blood type. People with type A blood get more malignant tumors that any other blood type.

ORIGINAL RESEARCH METHODS

Many copies of abstracts and articles in the Harvard Medical Research Library were read and cross-correlated to see if anything may have been overlooked.

FURTHUR FINDINGS

It was noted that Nitrogen levels were lowered in the main body, but excessive in the tumor masses. Also N-Acetylglucosamine and by-products of it's breakdown are excessive in patients with malignant tumors. N-Acetylglucosamine is the sugar chain that differentiates type A blood types from other types. There are 10 known sub-types of type A Blood. The sub-types of A inside the tumor is lower than that of the body's primary type A (That is A2 in the body, A6 inside the tumor.) Blodd types are determined by adding an anti-antigen to a sample and depending on the reactions the type is determined. (If Anti-A causes agglutination. The type is A; If Anti-B causes agglutination the type is B; If both cause agglutination the type is AB; if neither causes agglutination they type is O.)

QUESTIONS THAT AROSE AND RESPONSES FROM MEDICAL SOURCES

What would happen in Anti-A1 were injected into a patient with type O blood?

Nothing, as type O and B patients have Anti-A in their systems at all times in very low concentrations, but if it is used it is slow to be replaced.

What would happen if Anti-A1 were injected into a patient with type A or AB blood?

Treatment fatal - demise of patient imminent. At first it would be like a blood transfusion reaction in symptoms.

What would happen if Anti-A5 were injected into a patient with type A2 blood?

There may be a slight reaction, but there should be no harm to the patient.

What would happen to any cells or tumor with type A6 in them in a patient with type A2 blood if Anti-A5 were added to the patients blood?

The blood entering those cells would agglutinate and thus the cells would die due to lack of nutrients and oxygen.

ATTEMPT AT ACTIVE RESEARCH

We contacted a Dr. Jay Morjancouski (head of all Non-Human Primate research in the United States), and after explaining what we wanted to do with research animals, he advised that even if we could get the animals for research and if we could prove our theory 100% correct on them our test results would be considered null and void because we are dealing with components of the blood in animals which is different than human blood. A request for information of how we could test out theory was answered by "It must be tested on humans only".

PENDING CASE:Translated letter from Dr. Olivares

I was present at the autopsy of a clinical case of liver cancer. A Patient 47 years, male, country man, in which the clinical findings most important post mortem was hepatical cirrosis. Cause of his death. The patient blood type was O+, the blood on the sample from the hepatetical tissue was A+

Case of Study

A patient 36, years, female, intern in surgery with diagnosis of initial stomach cancer. The biopsy indicated Kultschitzky cells, with primary lesion in the lower abdominal, with intestinal metastasis. There were giant cellular anaplasia and mitosis presented. The presence of 5 A- antigen was presented The patient blood type was B+ The treatment consisted of: 1 day: 5cc of antiantigen A showed sever burning reaction. which decreased in a few minutes, injecting at a slower pace the antigen and antiantigen mixing with the patients own blood. that day the patient slept without any pain for the first time in many days. Second day: New I.V. injection of the antiantigen. There was no anaphylactic reaction. The patient claimed the pain was almost gone. Third day: New I.V. injection of the antiantigen, the patient gets out of bed. The appearance of puss through the fistula begins. There is no fever nor pain. Fourth day: Last antiantigen injection. The drainage of puss builds up. the patient claims to feel better. The doctors of the institution intervenes and close the fistula aggravating the infection. at no time was fever raised. Four days after the patient died. No autopsy was performed.